Women of reproductive age are frequently affected by the endocrinopathy known as a polycystic ovarian syndrome (PCOS). Since it was first described by Stein and Leventhal in 1935, this disease has been known to cause obesity, which accounts for at least 80% of ovulatory infertility. Obesity and PCOS are complicatedly related. Some women experience the first PCOS symptoms soon after they start their periods. According to my research, women with PCOS either create too much insulin or their insulin does not function as it should. As a result, insulin resistance is a frequent observation in PCOS. In women with PCOS compared to weight-contingent reproductively normal women, insulin action on skeletal muscle decreased by 35–40%, and this condition is made worse by obesity. Additionally, according to some research, up to 80% of women with PCOS are overweight or obese. Additionally, when weight increases, insulin resistance increases, and the level of circulating insulin rises, high levels of insulin are linked to obesity, making it possible to analyse the interplay between weight and insulin resistance. As a result, the level of testosterone will rise, which will disrupt follicular growth and development and result in polycystic ovarian morphology. There are so two approaches to connecting obesity and PCOS. I will first discuss how PCOS can induce obesity, and then I will discuss how obesity can cause PCOS. According to my investigations, women who are obese are more likely than the general population to develop PCOS because of insulin resistance. In the meantime, PCOS-afflicted women's obesity is exacerbated by rising androgen levels. The risk factors linked to an increased risk of PCOS in obese women include increased levels of luteinizing hormone (LH), hyperinsulinemia, and elevated levels of hyper-androgens. These risk factors also contribute to obesity in PCOS patients.